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These results confirm that incorporating PMA into standard DNA-based biocide assessment protocols is both feasible and beneficial

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4.3.4 Organelle dysfunction CS also impairs the function of critical organelles central to ferroptosis regulation: (1) Mitochondrial Damage: CS stimulates DRP1-dependent mitochondrial fission, resulting in fragmented, dysfunctional mitochondria that overproduce reactive oxygen species (ROS)

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193 These findings suggest that inhibiting ferroptosis may be a potential strategy for treating reproductive toxicity caused by drugs or stress