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Consequently, this leads to Ca 2 + dyshomeostasis and mitochondrial depolarization, cytochrome c release and apopotosome formation, permeabilization of the outer mitochondrial membrane as well as causing the hyperproduction and release of O 2 and H 2 O 2 leading to other free radical damage 70

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It also chelates iron, which accumulates abnormally in PD brains and contributes to oxidative damage, providing additional protection

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Cagrilintide acts on calcitonin-related receptors that can influence some glucagon-adjacent signaling, but through a different mechanism