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Based primarily on mouse and isolated human cell studies, this myelofibrosis is thought to result from the accumulation of platelet precursor cells in the bone marrow and their release of excessive amounts of cytokines that stimulate bone marrow stromal cells to become fiber-secreting fibroblasts and osteoblasts

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Currently, clock gene expression can be studied in vivo in the peripheral blood, oral mucosa, and hair follicles [13]

Severe vascular toxicity associated with cisplatin-based chemotherapy

Further research demonstrates neuroprotective potentials of CB 2 receptor in PD