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A recent breakthrough demonstrated targeted mtDNA-editing in human cells by co-delivering a DNA end-joining system and site-specific mitochondrial nucleases, opening new possibilities for direct mitochondrial genome engineering (Fu et al, 2025)

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In a publication by Harman, he explored the hypothesis suggesting that free radicals might have a notable impact on the aging process

Interestingly, an electrophilic molecule, 15-deoxy-12,14)-prostaglandin J2, is produced by convoluted blood flow with shear stress and activates Nrf2 in endothelial cells [23]