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Such modifications can result in loss of enzymatic activity, altered cellular signaling, and degradation of structural proteins, contributing to cellular dysfunction and death [36, 37]

Hashemi Tari, S., Sohouli, M

On the other hand, several other groups reported that rat in vitro spermatogenesis progressed no further than the pachytene spermatocyte stage 24,25,26,27

Current pharmacological interventions for oxidative stress in the nervous systemsuch as edaravone (15, 16) and N-acetylcysteine (16, 17)act on single targets and employ relatively narrow mechanisms, and are associated with dose-limiting adverse effects and loss-of-benefit phenomena (e.g., end-of-dose deterioration and motor complications), rather than durable disease modification (18)

Colorectal cancer (CRC) is caused by continuous stimulation of the signaling pathway of the KRAS protein