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FFAs increase ROS levels in monocytes dose-dependently, leading to adhesion of the monocytes to the endothelial cells [38]a critical mechanism in the development of atherosclerosis [39]

Further complicating this picture is the crucial concept of redox hormesis, which reveals that the biological response to ROS follows a non-linear, often U-shaped, curve

doi: 10.4049/jimmunol.180.6.4299 327 Vieira-NetoA.GalvoK

As the trans-octadecenoate of azacitidine, the cellular uptake was substantially less dependent on the nucleoside transporters, and it has better membrane permeability