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Secondary iron overload is associated with genetic factors and acquired diseases [29]

The mechanism of epilepsy induced by the imbalance of the CLC channels or gene mutation may be related to its regulation of excitability of the cell membrane and the transport functions of electrolytes, water, and nutrients (Wei et al., 2017)

[11] found that several single nucleotide polymorphisms (SNPs) in the GPx3 gene promoter region, which decreased the expression of this enzyme [12], were associated with an increased incidence of ischemic stroke before the age of 45

Cell surface biliverdin reductase mediates biliverdin-induced anti-inflammatory effects via phosphatidylinositol 3-kinase and Akt

The results exceeded what either approach achieved on its own