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162 These metabolic alterations eventually lead to the overproduction of HA and Plasminogen activator inhibitor 1 (PAI-1) and the expansion of Tregs and myeloid-derived suppressor cells (MDSCs) populations

STAT3/HIF-1 signaling activation mediates peritoneal fibrosis induced by high glucose

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Unusually, gliT expression was shown to be independent of the overall gli cluster, as it could be specifically induced in A

A 9-amino acid cyclic peptide, CAR (sequence CARSKNKDC) was particularly effective in homing to wounds and shown to recognise angiogenic blood vessels in regenerating tissues 3