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Hepatology 30(1):186195 Win S et al (2021) Hepatic mitochondrial SAB deletion or knockdown alleviates diet-induced metabolic syndrome, steatohepatitis, and hepatic fibrosis

In this respect, the diminution of Se activity as an antioxidant can explain the elevated lipid peroxidation in infertile patients

Together, our data suggest that p53 controls a stress-responsive gene programme, rather than a single downstream gene, that facilitates fasting-dependent recruitment of macrophages into eWAT under metabolic stress