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Again, factors that may have led to the high level of heterogeneity between studies include fasting status, differing laboratory methods and varied selection of controls

doi:10.1111/j.1468-3083.2005.01248.x

Hershko, A., Heller, H., Elias, S

While these models do provide some degree of insight into the pathophysiologic basis for cardiometabolic and neurodegenerative diseases, they do not completely replicate the complexity, in terms of immune diversity and microbiome diversity, of those diseases in humans (25, 28)

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