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Instead of accurately interpreting sensory input, pain-processing circuits become hyperresponsiveturning normal movement, pressure, or minor inflammation into persistent pain

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ITalk labeling in Raw264.7 cells, followed by enrichment and Western blot detection, confirmed the itaconation of both proteins (Fig

Once released into the extracellular space, especially after tissue injury, they undergo changes from reducing to oxidizing environments that alter their function, associated with mitochondrial dysfunctions

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