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Poor brain function in people with liver failure (hepatic encephalopathy)

UA administration protected against the cisplatin-induced depletion of the renal GSH pool, the inhibition of GPx and superoxide dismutase (SOD) activity, and a decrease in the gene expression of NADPH oxidase 2, which was the main source of oxidative stress in this model

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Human-Specific Mechanism Involving a Non-human glycan in Red Meat 2.3.2.1