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A meaningful proportion of the weight lost on GLP-1 therapy is fat-free mass (skeletal muscle, organ tissue, water) rather than fat alone

This occurs through the direct impact of the peptide on body fat tissue, whereby it activates the beta-3 adrenergic receptor(s) involved in the fat-burning process within the body

The implications extend beyond obesity and diabetes to every class B GPCR target in the human genome, suggesting that many receptors previously considered "undruggable" by small molecules may be amenable to similar approaches

The JAK-STAT pathway regulates CD38 on myeloma cells in the bone marrow microenvironment: therapeutic implications

Whats the reality of bringing up stubborn calves with DES