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Compared with diabetic rats, the left ventricular hemodynamic parameters and myocardial ultrastructure changes (myocardial hypertrophy and fibrosis) are obviously normalized in diabetic animals treated with NaHS, which may be associated with upregulation of cardiac myosin light chain kinase (MLCK) and downregulation of serum creatine kinase MB isozyme (CK-MB) (Yang et al., 2016a)

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Therefore, timely inhibition of lipid metabolism disorders, the inflammatory response, and VSMC migration is the focus of atherosclerosis prevention and treatment of [8,9,10,11,12]

Each tag requires a specific cofactor (vitamin, mineral, or amino acid) to be available