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PLoS Biol 16:e2006203 Friedmann Angeli JP, Schneider M, Proneth B, Tyurina YY, Tyurin VA, Hammond VJ, Herbach N, Aichler M, Walch A, Eggenhofer E, Basavarajappa D, Radmark O, Kobayashi S, Seibt T, Beck H, Neff F, Esposito I, Wanke R, Forster H, Yefremova O, Heinrichmeyer M, Bornkamm GW, Geissler EK, Thomas SB, Stockwell BR, ODonnell VB, Kagan VE, Schick JA, Conrad M (2014) Inactivation of the ferroptosis regulator Gpx4 triggers acute renal failure in mice

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Upon further dosing of mice with dabrafenib and trametinib, without ferrostatin-1, the GPX4 WT tumours relapsed and the GPX4 KO tumours did not (Fig

In addition, further experiments found that in Arabidopsis wounding-triggered Ca 2+ signaling promoted the biosynthesis of JA (a potential regulator of defense response) by stimulating lipoxygenase and/or inhibiting phosphorylation of repressors JAV1 within the JAV1-JAZ-8-WRKY51 complex, finally activating plant defense response (Mousavi et al., 2013