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Description

69 LiY.XingM.YangQ.WangY.JiangJ.ZhaoY.et al

Member (20032015) Indiana University School of Medicine Vice-Chair of Clinical Practice, Department of Neurology Development (20032005) Indiana University School of Medicine Department of Neurology Clinical Practice Committee (incentive plan, coding, compensation, service standards)

Consistent with the results observed in in vivo experiments, GHK-Cu significantly restored Nrf2 expression in the cytoplasm and reduced it in the nucleus while significantly reducing Keap1 levels in the cytoplasm compared with the CSE-exposed cells without GHK-Cu (Figures 6DF)