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The key pathological hallmarks of AD include amyloid-beta (A) plaque accumulation, neurofibrillary tangles of hyperphosphorylated tau, chronic neuroinflammation, oxidative stress, and mitochondrial dysfunction (Lv et al., 2020)

The release of mitochondrial DNA (mtDNA), recognized as damage-associated molecular patterns, can activate the cGAS-STING1 and NF-B pathway and induce pro-inflammatory factor expression

Protection of germ-free mice from infection by Helicobacter felis after active oral or passive IgA immunization

Can taking bromelain or papain help with low back pain in people with osteoarthritis

Supplementation may cause accumulation and oxidative paradoxhigh antioxidant levels can promote pro-oxidant effects in these contexts