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10.3390/ijms19092549 21 BrodinH

Although there is an unresolved dispute over the contribution of the tight junction barrier to the disease pathology, recent studies have concluded that even in the presence of a normal underlying immunity, functional abnormalities of tight junction proteins could result in these diseases

136(2):548S-552S

Amelioration of hepatotoxicity by biocleavable aminothiol chimeras of isoniazid: design, synthesis, kinetics and pharmacological evaluation

[207] showed that A 142 exposure induces astrogliosis, A 2A Rs upregulation, and impaired GLT-1 glutamate transporter function and glutamate uptake in cultured astrocytes, effects that could be blocked by A 2A R antagonist SCH 58261