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A by-product of thiol oxidation is peroxides, which damage the cell

LY46 had more upregulated genes under control conditions at 72 h, followed by PEG treatment at 72 h (Fig

1a), and all treatments lead to decreased cell viability, as measured by annexin V-PI assay (Fig

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Although GSH extrusion may provide a simple mechanism to circumvent the normally protective functions of GSH, there is increasing evidence that GSH export is required for activation of specific apoptotic signaling pathways and/or for proper dismantling of cellular components (Coppola and Ghibelli, 2000