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This latter induced the formation of reactive oxygen species (ROS) in the hepatocytes resulting in the increase in cellular thiobarbituric acid reactive substances (TBARS) and glutathione (GSH) depletion

New insights into the pathogenesis of non-alcoholic fatty liver disease: gut-derived lipopolysaccharides and oxidative stress

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HDL and apoA-I were demonstrated to suppress CD11b expression on human monocytes through both ABCA1-dependent and independent mechanisms (814)

Alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptors mediate excitotoxicity in the oligodendroglial lineage