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Thats mainly because the GGT enzyme in the intestinal tract degrades it before it can be used

Growing evidence identifies mitochondrial dysfunction, including abnormalities in mitochondrial DNA, impaired bioenergetics, disrupted quality control, and redox imbalance, as a central pathological feature of MDD

PD-1 can upregulate the expression of PD-L1 in tumor cells and ultimately inhibit the proliferation of T cells and reduce immune function by binding to its ligand PD-L1, thereby facilitating tumor cell immune escape [61,62,63,64], this is the mechanism by which tumor cells evade immune cells with the help of PD-1 in the absence of glutamine

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