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Recent hypotheses suggest that GIP induces appetite suppression through independent receptors in the brain, mitigates gastrointestinal side effects of GLP-1 action, and prevents desensitization of GLP-1 receptors, thereby acting as a biased GLP-1RA [87]

Our results are consistent with similar studies reported by other investigators (Klongpanichapak et al ., 2006

Similarly, hyperphosphorylated Tau, a pathological hallmark of AD, promotes cerebral iron deposition, creating a vicious cycle in AD progression (Wang F