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FIGURE 1 2.1 Epigenetic regulation and chromatin remodeling Epigenetic alterations constitute a critical factor driving both the induction and maintenance of the reversible DTP phenotype ( 2.2 Transcriptional plasticity and gene regulatory networks Reversible DTP formation is closely associated with transcriptional rewiring, enabling cells to adapt to therapeutic stress temporarily ( 0 /G 1 arrest and metabolic rewiring that supports survival under cytotoxic stress ( 2.3 Metabolic reprogramming as an adaptive response Reversible metabolic adaptations critically support the persistence phenotype ( + cell populations ( Elevated OXPHOS activity not only supports reduced proliferation rates but also limits reactive oxygen species (ROS) accumulation, thereby protecting DTP cells from oxidative stress-induced death ( 2.4 Influence of the tumor microenvironment (TME) The reversibility of the DTP state is also regulated by dynamic interactions with the tumor microenvironment

Detection and quantification of superoxide formed within the periplasm of Escherichia coli

Dubois N, Crompot E, Meuleman N, Bron D, Lagneaux L, Stamatopoulos B

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By neutralizing oxygen species and free radicals glutathione helps preserve integrity and function