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Human peripheral blood mononuclear cells (PBMCs) are unaffected at concentrations of elesclomol that have a significant killing effect on cancer cells, and elesclomol cannot induce copper ion enrichment in PBMCs (212)

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The mutations in this gene, which enhance oxidative stress in the cells, promote protein deposition, disrupt intracellular calcium ions, and cause the diffusion of toxicity, are responsible for around 20% of familial ALS and 2% of sporadic ALS [24]

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