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Ferroptosis is accompanied by iron overload, excess lipid peroxidation and a large amount of ROS production, which can accelerate a collection of pathological processes such as inflammation, mitochondrial dysfunction, ERS, and OS that further lead to the increase of CD and damage to the normal function of cardio-cerebral vessels

GSSG is then recycled back to GSH by GSH-reductase using NADPH as cofactor and the ratio of GSH to GSSG regulates redox dependent cell signaling

The activation of these events results in an increase in vasoconstriction and the promotion of thrombus formation, which can ultimately lead to the development of atherosclerosis (16)

Macrophages (2 10 5 ) were incubated in vitro with tBHP (40 M) or tBHP plus A

Seven measures of antioxidant status were determined using Trolox equivalent antioxidant capacity, uric oxidase, alpha-tocopherol, total glutathione, cytosolic glutathione peroxidase, and superoxide dismutase