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Both peptides may facilitate accelerated recovery from muscle strain injuries and improve healing quality following muscle tissue damage

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KRAS can activate downstream factors through phosphorylation reactions, and ultimately promote cell proliferation by regulating signaling pathways such as the RAS-RAF-MEK-ERK and PI3K-AKT pathways 42

It is recently reported that fatty acid synthase (FAS) inhibition triggered caspase-dependent apoptosis and suppressed the expression of c-MET in DLBCL cell lines [32]

In comparison with the SA suspension, SA-SMEDDS showed significant prolonged T max , t 1/2 , and MRT after oral administration