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Initially a well-defined condition, it was soon linked to numerous nonspecific symptoms despite a lack of solid evidence that it could provide a satisfactory explanation for such patients [1]

Oxidative stress is another key factor, whereby the accumulation of ROS damages cellular components, particularly the mitochondria

As president and correspondent of our Class of 1952, I appreciate the continuing engagement of many of our classmates in supporting the class and Cornell, and in sharing their news of this phase of our lives

Blockade of Myeloid-Derived Suppressor Cell Expansion with All-Trans Retinoic Acid Increases the Efficacy of Antiangiogenic Therapy

Bellinger DL, Millar BA, Perez S, Carter J, Wood C, ThyagaRajan S, et al