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The formation of DHA happens through spontaneous oxidation from MDHA

Both physiological and pathological stimulations were performed

It significantly reduces systemic estradiol (E 2 ) and vascular endothelial growth factor (VEGF) levels, selectively inhibits the VEGF receptor-2-driven PI3K/Akt/mTOR signaling pathway within ectopic lesions, suppresses pathological neovascularization, and reactivates intrinsic apoptosis through cytochrome c release and caspase-3 cleavage, thereby disrupting the hormonal, angiogenic, and survival pathways required for EMs persistence ( in vivo and in vitro studies have shown that Rg3 also has a strong anti-fibrotic effect