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123 A recent study similarly highlighted the contribution of gut microbial dysbiosis to perturbing bile acid dysmetabolism in individuals with cancer cachexia, especially the reduction in the levels of several secondary bile acids, mainly taurodeoxycholic bile acids, which occur before the development of cachexia

These findings raise the question about the mechanism of PI3K signaling activation by androgens

More detailed mechanisms are yet to be widely researched

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In a mouse model, TRP deficiency induced gut microbiota dysbiosis, altered the formation of various gut metabolites and expression of regulatory T-lymphocytes, and increased proinflammatory cytokine levels [128,129]