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Several autistic models showed an increase in neuronal apoptosis resulting from the decrease in the expression of Bcl-2/Bcl-2-associated X protein (Bax) that increases activation of caspase-3, boosting uncontrolled apoptosis and eventually resulting in ASD

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However, the in vitro co-culture assay did not detect a reduction in the number of neural progenitors (Fig

On rare occasions, small intestinal illness may be the secondary cause of this deficiency (190)

However, on the TNF flow cell surface, all dosed subjects in cohort 7 showed increasing signal compared to baseline which was confirmed with the anti-human IgG reagent, but not the AMG 966-specific antibody 1A3, indicating these subjects developed endogenous anti-TNF antibodies as a result of AMG 966 treatment ( Figures 8F, H )