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RS induces cellular senescence, which hampers cell reproduction and proliferation in response to damage, and senescent cells develop a phenotype characterized by the secretion of factors like interleukins, chemokines, matrix metalloproteases (MMPs), and extracellular matrix (ECM) proteins [15]

Since supraphysiological levels of NADH were reported to inhibit NAD + -dependent sirtuin deacetylase (SIRT17) (50), accumulated NADH in mitochondria repressed mitochondrial SIRT3 activity leading to hyperacetylation of mitochondrial proteins (42, 47) (Fig

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Icariin alleviates ferroptosis-related atherosclerosis by promoting autophagy in xo-LDL-induced vascular endothelial cell injury and atherosclerotic mice