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This study shows that tumour tissue of origin and oncogenic drivers combine to regulate glutamine metabolism

Docking accelerates the identification of potential therapeutics by modeling intermolecular interactions and optimizing complementarity and structural precision

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Mutation-independent therapies can have various downstream targets that aim to improve mitochondrial respiration, reduce mitochondrial stress, inhibit or delay RGC apoptosis, and/or promote RGC survival

In particular, PDAC cells require Gln, and supplementation of aKG with nonessential amino acids (NEAAs) rescues PDAC cells from Gln deprivation-mediated growth inhibition 11,26 , supporting the present data showing that Gln is a critical source for aKG production