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87.6%) and lower 1-year mortality rates (15.5% vs

These include both enzymes, such as superoxide dismutase (SOD), catalase, glutathione peroxidase, glutathione reductase, thioredoxins, peroxiredoxins, and non-enzymatic antioxidants such as glutathione, vitamin C, and E (Quijano et al., 2016

soft-tissue repair) and TB-500 (Thymosin Beta-4 fragment

The overexpression of critical autophagic genes exhausts the levels of the mutant p53 protein, and treating cells with autophagy inhibitors increases the stability of mutant p53

The intersection of DNA damage response and Ferroptosis-A rationale for combination therapeutics