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Unexpected activation of the Akt-mTOR pathway augments the transcription of gluconeogenesis-related metabolic enzymes, including GLUTs and HK2, which elicits a series of cascade reactions to increase ABC transporter proteins expression, proliferation and antiapoptotic capacity of leukemic cells, leading to the MDR phenotype in leukemic cells (Zhang X

Vilstrup H, Amodio P, Bajaj J et al (2014) Hepatic encephalopathy in chronic liver disease: 2014 practice guideline by the American Association for the Study Of Liver Diseases and the European Association for the Study of the Liver

Carnosine dipeptidase II (CNDP2) has been described to support the catabolism not only of cysteinylglycine 57 but also of other dipeptides, such as those containing glutamine 35

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