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1 Introduction OSAHS is primarily characterized by the recurrent partial or complete obstruction of the upper airway during sleep, leading to chronic intermittent hypoxemia and associated pathophysiological consequences, including disrupted sleep architecture, prolonged sympathetic activation, and carbon dioxide retention (1)

Correlation between T2 relaxation time and intervertebral disk degeneration

Metabolic syndrome was defined according to the criteria of the national cholesterol education program

Cortisol / prolactin & unknown long-term profile: A modest rise in cortisol and prolactin can occur

Due to the rapid proliferation of GBM, the TME is suppressed by factors such as acidity and hypoxia, rendering immunotherapy ineffective [95, 96]