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Unlike PqsA, ArqI lacks tryptophan residues and therefore would have no tryptophan emission

This cascade also appears to intersect with mitochondrial and ER stress pathways, where redox imbalance and unfolded protein responses converge-mechanisms mechanistically reminiscent of those implicated in neurodegeneration

This ATP deficiency presumably results in a reduced capacity to maintain the red cell membrane and diminished erythrocyte deformability, resulting in a shortened lifespan and destruction in the spleen

2017;2017 doi: 10.1155/2017/3140673

The physiological role of the ribulose monophosphate pathway in bacteria and archaea