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On one hand, NADH is overproduced due to overnutrition and hyperglycemic activation of the polyol pathway [81, 103]

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Acta 1852 (2), 252261

Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]