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Ultimately, our proposed paracrine mechanism would enable rapid, dynamic coordination of intra-islet DA signaling to further fine-tune regulation of insulin and glucagon secretion

demonstrated that polymorphonuclear MDSCs (PMN-MDSCs) in the TME are susceptible to ferroptosis due to hypoxia-mediated downregulation of GPX-4 and accumulation of AA resulting from upregulation of their own FA transporter protein 2

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The datasets used and analyzed during the present study are available from the corresponding author upon reasonable request