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At 1 h after EpoB-induced regeneration, the fluorescence lifetime at the axon tip was significantly longer than at the axon shaft of the same axon (3.64 ns at the axon tip versus 3.24 ns at the axon shaft), indicating increased membrane tension at the regenerating site (Fig

Cholesterol is essential for maintaining intracellular homeostasis [55], and elevated cholesterol levels within tumor cells play a crucial role in enabling evasion of immune surveillance and fostering the upregulation of inhibitory immune checkpoint genes, consequently impeding the efficacy of antitumor responses [56]