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Most important, total LOXs activity was strikingly higher in the aortic rings of old WT mice than in those from both young WT mice and old LOXL2+/ mice, as evidenced by a markedly higher level of BHZ incorporation

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This model explains why mutation of either site abolishes Src activation in the minimal in vitro system, where cooperative rebinding is essential, while single-site mutants retain partial activity in cellular contexts, where additional scaffolding interactions and membrane confinement can partially compensate