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Description

Cell 173 , 7489.e20 (2018)

A Potent And Specific CD38 inhibitor ameliorates age-related metabolic dysfunction by reversing tissue NAD+ decline

In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]