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In this aspect, it suggested that dysfunction of the mitochondria and oxidative stress might be the main factor behind the T-2 toxin-induced apoptosis in the fetal brain [105]

10.1002/cbic.201500589 82 YinY.SasakiS.TaniguchiY

Treatment of primary cultures of human skin keratinocytes or HaCaT cells with antisera against rGRP blocked the effects of rGRP protein on wound closure and repair process