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10.3329/jafmc.v14i1.42723 140 ToprakB.SollederH.Di CarluccioE.GreensladeJ

The primary theoretical concern is that sustained NAD+ elevation through chronic NNMT inhibition could activate pathways (PARP, SIRT1) that have context-dependent roles in cancer biology NAD+ supports DNA repair and cellular longevity, but also fuels rapidly dividing cells

These molecules target metabolic processes in liver, heart, muscle, and WAT through multiple signaling pathways (48)

They are better understood as complementary research tools than as alternatives

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