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Knockout of the enzyme which synthesizes GPE, phosphatidylserine decarboxylase Psd1 (mammalian Pisd), in yeast and worm models of PD was shown to result in the formation of -syn foci, ER stress, defects in trafficking, and decreased respiration (Wang et al., 2014)

Accordingly, SHMT inhibition has recently been identified as a major therapeutic vulnerability in T-ALL [16, 17]

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Furthermore, evidence has revealed that the inhibition of caspase-2 prevents hyperlipidemia and reduces hepatic steatosis, liver apoptosis, and mitochondrial mitophagy (190, 194)