After a marked decline in the mid-twentieth century, incidence has risen again over the last two decades, notably in high-income countries, reshaping the daily landscape of infectious neurology and reviving the clinical relevance of neurosyphilis as the great imitator ( Autoimmune encephalitis is now a well-defined, treatable cause of subacute neuropsychiatric syndromes, supported by consensus diagnostic criteria that have sharpened clinical recognition and encouraged early immunotherapy when appropriate ( Against this backdrop, a small but increasingly cited body of reports has described neurosyphilis presenting with a limbic encephalitis phenotype and, more provocatively, cases in which neurosyphilis coexists with neuronal surface antibodies, including anti-NMDAR antibodies ( Methods Because this phenomenon is uncommon and largely captured through case-based evidence, we performed a structured narrative review focused on publications describing neurosyphilis with a limbic encephalitis phenotype and publications reporting neuronal surface antibodies in the context of neurosyphilis
-Alanine was found to interact with and displace [3H]glycine at the strychnine-insensitive glycine site on the NMDA receptor [60]
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Mitigation of heavy metal stress in maize ( Zea mays L.) through application of silicon nanoparticles
Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-kB) is a complex of transcription factors responsible for expression of many inflammatory genes
