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The dysfunctional molecules created by glycation are termed advanced glycation end products (AGEs) (Sugiyama 1996

In addition, dichloroaceetate treatment alone caused ER stress to an extent that was nearly as robust as the bona fide ER stressor TM, as seen by upregulation of UPR target genes (Figure 7B) and splicing of Xbp1 (Figure 7C)

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