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Namely, in analogy, we evidenced the occluded superior sagittal sinus-produced central disturbances (i.e., intracranial (superior sagittal sinus) hypertension) rapidly transmitted to the periphery (i.e., portal, caval hypertension, aortal hypotension, multiorgan lesions) [20]

Few studies have evaluated the interaction between the delivered cells and the native disc microenvironment, especially with regard to the innervated disc

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In the present study we sought to expand this concept by determining if shunting glutamate from the glutathione cycle can shape excitatory neurotransmission

AMA Drug Evaluations Annual 1991