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We generated three constructs, the full-length ElpQ lacking the native signal sequence (aa 19-343), along with N-terminal (aa 19-206) and C-terminal (aa 168-343) truncation constructs that were termed ElpQ FL, ElpQ N, and ElpQ C, respectively

By integrating findings from animal models, clinical imaging, and molecular biology, this work elucidates how dysfunctions in GABAergic interneurons (e.g., PV+, SST+, VIP+), GABA signaling pathways, and associated genes collectively disrupt the E/I balance in the brain, leading to core ASD symptoms such as social communication deficits, repetitive behaviors, and sensory abnormalities

doi: 10.1021/acschembio.9b00939 231 BiJYangSLiLDaiQBorcherdingNWagnerBAet al

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