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In the absence of functional SLC30A10 proteins (either by knocking out the gene or introducing a loss-of-function mutation), Mn accumulates in Golgi-derived vesicles, ultimately leading to cytotoxicity 11

In the case of proteins that have a nearby cysteinyl residue, a disulfide bond forms between the two sulfur atoms

In parallel, supervised analysis was performed on the same dataset and on the remission case control study ( n = 17) defining cell subsets based on canonical markers and followed the scheme illustrated in Supplementary Fig

Enhancing NAD + salvage metabolism is neuroprotective in a PINK1 model of Parkinson's disease

The buffer you drew earlier is what gets pushed out here