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In hepatic cells, phosphorylated HSP27 promotes autophagy and lipid clearance, suggesting its therapeutic potential in MASLD and non-alcoholic steatohepatitis (NASH), now referred to as metabolic dysfunction-associated steatohepatitis (MASH)

Subsequently, studies conducted in vitro indicate that defective proteasome activity disturbs A degradation consistent with accumulation of this neurotoxic peptide and plaque formation (352)

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Glutamine and barrier function in cultured caco-2 epithelial cell monolayers